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| Outcompeting p53-mutant cells in the normal esophagus by redox manipulation D Fernandez-Antoran, G Piedrafita, K Murai, SH Ong, A Herms, C Frezza, ... Cell stem cell 25 (3), 329-341. e6, 2019 | 99 | 2019 |
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| p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition K Murai, S Dentro, SH Ong, R Sood, D Fernandez-Antoran, A Herms, ... Nature Communications 13 (1), 6206, 2022 | 19 | 2022 |
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| The single-progenitor model as the unifying paradigm of squamous epithelial maintenance G Piedrafita, V Kostiou, A Wabik, B Colom, D Fernandez-Antoran, ... BioRxiv, 716639, 2019 | 5 | 2019 |
| Precancer: Mutant clones in normal epithelium outcompete and eliminate esophageal micro-tumors B Colom, A Herms, MWJ Hall, SC Dentro, C King, RK Sood, MP Alcolea, ... bioRxiv, 2021.06. 25.449880, 2021 | 2 | 2021 |
| The nucleotide kinase Nadk is required for ROS detoxification and constitutes a metabolic vulnerability of NOTCH1-Driven T-ALL E De Braekeleer, J Hsu, C Hervieu, K Tzelepis, S Indraccolo, AJ Warren, ... Blood 132, 2615, 2018 | 2 | 2018 |
| Levelling out differences in aerobic glycolysis neutralizes the competitive advantage of oncogenic A Herms, B Colom, G Piedrafita, K Murai, SH Ong, D Fernandez-Antoran PIK3CA, 0 | 2 | |
| Epithelioids: Self-sustaining 3D epithelial cultures to study long-term processes A Herms, D Fernandez-Antoran, MP Alcolea, A Kalogeropoulou, ... bioRxiv, 2023.01. 03.522589, 2023 | 1 | 2023 |
| S857 THE NUCLEOTIDE KINASE NADK CONSTITUTES A METABOLIC VULNERABILITY OF NOTCH1‐DRIVEN T‐ALL E De Braekeleer, M Gu, C Hervieux, J Ino Hsu, D Aspris, E Abby, ... HemaSphere 3, 382-383, 2019 | 1 | 2019 |
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| S858 SUZ12 INACTIVATION COOPERATES WITH JAK3 MUTANT SIGNALING TO DRIVE T-ALL DEVELOPMENT M Broux, C Prieto, S Demeyer, MV Bempt, L Alberti-Servera, R Vandepoel, ... HemaSphere 3 (S1), 383-384, 2019 | | 2019 |
Swee Hoe OngWellcome Sanger InstituteVerified email at sanger.ac.uk
